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Richard R. Fay, DC

Omega-3s and Brain Injury Recovery


Even mild head injuries can damage brain cells and increase oxidative stress. This can disrupt normal cell metabolism and cause further cell damage. Research indicates that higher blood cell levels of EPA and DHA fatty acids can help inhibit cell death, reconnect damaged neurons, activate genes that repair damaged cells, and turn off genes that promote brain inflammation.

Increased DHA levels inhibit neuronal cell death and is an important neuro-protective agent.

DHA is incorporated into the phospholipids in neuronal membranes, which in turn can influence not only the membrane chemical and physical properties but also the cell signaling involved in neuronal survival. Studies have indicated that DHA supplementation inhibits neuronal cell death under challenged conditions, supporting a notion that DHA is an important neuroprotective agent.

Low dietary Omega-3 consumption responsible for up to 96,000 preventable deaths each year.

A recent study by Harvard School of Public Health revealed that Omega-3 fatty acid deficiency is the sixth biggest killer of Americans- even more deadly than excess trans fat intake. The study utilized 2005 data from the US National Health Center for Health Statistics and revealed there are between 72,000 and 96,000 preventable deaths each year due to Omega-3 deficiency.

High EPA and DHA blood levels could reduce the oxidative stress developed in brain due to TBI/concussion

The role of Omega-3s in the neuronal growth and maintenance is well documented. Omega-3 intake could lessen the secondary effects of the trauma in brain. Omega-3 fatty acids (FAs) could reduce the oxidative stress developed in brain due to TBI. The inclusion of Omega-3 FA in diet could normalize the levels of brain-derived neurotrophic factor (BDNF), and thus, it could restore the survival of neuronal cells.

Increased DHA levels inhibit neuronal cell death and is an important neuroprotective agent

DHA is incorporated into the phospholipids in neuronal membranes, which in turn can influence not only the membrane chemical and physical properties but also the cell signaling involved in neuronal survival. Our studies have indicated that DHA supplementation inhibits neuronal cell death under challenged conditions, supporting a notion that DHA is an important neuroprotective agent.

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